Principles and illustrations of morbid anatomy : adapted to the elements of M. Andral, and to the cyclopaedia of practical medicine, being a complete series of coloured lithographic drawings, from originals by the author : with descriptions and summary allusions to cases, symptoms, treatment, &c. : designed to constitute an appendix to works on the practice of physic, and to facilitate the study of morbid anatomy in connexion with symptoms / by J. Hope.

  • Hope, James, 1801-1841.
Date:
1834
Catalogue details

Licence: Public Domain Mark

Credit: Principles and illustrations of morbid anatomy : adapted to the elements of M. Andral, and to the cyclopaedia of practical medicine, being a complete series of coloured lithographic drawings, from originals by the author : with descriptions and summary allusions to cases, symptoms, treatment, &c. : designed to constitute an appendix to works on the practice of physic, and to facilitate the study of morbid anatomy in connexion with symptoms / by J. Hope. Source: Wellcome Collection.

Provider: This material has been provided by the Francis A. Countway Library of Medicine, through the Medical Heritage Library. The original may be consulted at the Francis A. Countway Library of Medicine, Harvard Medical School.

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    B, from inadequate sangiiilication; C, from atrophy of the lung, observed principally in the aged. Lesions of Nutrition, These are situated, according to the foregoing principles, either in the walls of the air-vesicles, or in the intervening cellular tissue. They consist (in the lungs, as in every other organ) of 1. an increase, 2. a diminution, or, 3. a perversion of the natural nutrition. I. Increased Nutrition, or Hypertrophy.—Of this there are two forms : A, consisting of an augmentation of the number and size of the air-vesicles, without thickening of their walls,—con- sequently, without induration, but with amplification of the pulmonary substance. Here must be ranged a variety in which some of the vesicles and capillary bronclii are thickened as well as dilated, constituting one of the forms of pulmonary emphysema. B, consisting of thickening of the vesicular walls, with diminution or obliteration of their cavities,—consequently, with induration of the pulmonary substance. This form may occupy either large portions of the lung, or only single lobules, or even vesicles. In the latter case, according to the views of Andral, it constitutes the pulmonary granulations of M. Bayle. Hypertrophic induration sometimes affects the interlobular cellular tissue, either separately, or in connexion with induration of the lobes. II. Diminished Nutrition, or Atrophy.—In this there is a decrease in the number of air-vesicles, and an attenuation of their walls, often causing them to burst into, and coalesce with each other. Enlargement of the pulmonary vesicles from this coalition, no less than from the dilatation of each, before alluded to, con- stitutes pulmonary emphysema. HI. Perverted Nutrition.—This consists in a transformation of the cellular, into fibrous or cartilaginous tissue. Very rarely an osseous matter forms the vesicular walls.