Two cases of symmetrical necrosis of the cortex of the kidneys associated with puerperal, eclampsia and suppression of urine / by R. Jardine and J.H. Teacher.

  • Jardine, Robert.
Date:
[1911?]
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Licence: In copyright

Credit: Two cases of symmetrical necrosis of the cortex of the kidneys associated with puerperal, eclampsia and suppression of urine / by R. Jardine and J.H. Teacher. Source: Wellcome Collection.

Provider: This material has been provided by The University of Glasgow Library. The original may be consulted at The University of Glasgow Library.

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    only the structures which are known to be principally affected by these conditions, but also the glomeruli and interstitial tissues. To explain it some other factor must be brought in, and this was undoubtedly the thrombosis in the smaller renal arteries. Haemor- rhage, weakness of the circulation, and the puerperal state may all have helped to bring about the thrombosis, but we are of opinion that the ultimate cause of it was spasmodic contraction of the renal vessels. This gives the most satisfactory explanation of the symmetry of the lesion. REFERENCES. Allan, G. A., and G. Glasg. Med. Journ., 1910, vol. Ixxiv. p. 25. Haswell Wilson Bradford, Rose, and Journ. Path, and Bacterial., Cambridge, 1898, Lawrence vol. v. p. 197. Griffith and Herringham Ibid., 1906, vol. xii. p. 237. Jardine, R Journ. Obst. and Gyncec. of British Empire, London, 1906, vol. x. p. 32. Klotz, Oscar .... Am. Journ. Obst, N.Y., 1908, vol. lviii. p. 619. Lloyd, H. C Lancet, London, 1906, vol. i. p. 146. Osler in Osier and McCrae's System of Medicine, London, 1909, vol. vi. p. 625. Schmorl Puerperal Eclampsia, Leipzig, 1893. Weber, Parkes .... Lancet, London, 1909, vol. i. p. 601. DESCRIPTION OF PLATES. Plate XX. Fig. 1.—The kidney from Case 1, showing the irregular distribution of the necrosis (infarction). Cf. text, p. 139. Plate XXI. Fig. 2.—The kidney from Case 2, showing the extremely complete and regular necrosis (infarction) of the cortex. Plate XXII. Fig. 3.—Thrombosed artery and vein from the edge of the infarct in Case 1, showing the short, dense amorphous plug in the artery and thrombus of red or mixed character in the vein. There is a great deal of hemorrhage between the tubules, which are nearly all necrotic. ( x 80.) Fig. 4.— Thrombosed artery from the edge of the infarct in Case 2. Note how sharply defined the infarction is, but the position of its margin is similar to that in Case 1. The vein is not recognisable. Most of the red thrombus has fallen out of the distal portion of the artery. ( x 80.)