Visceral angioneuroses : (the visceral crises of vasomotor ataxia) / by Solomon Solis Cohen.

  • Solis-Cohen, Solomon, 1857-1948.
Date:
1910
Catalogue details

Licence: In copyright

Credit: Visceral angioneuroses : (the visceral crises of vasomotor ataxia) / by Solomon Solis Cohen. Source: Wellcome Collection.

Provider: This material has been provided by The Royal College of Surgeons of England. The original may be consulted at The Royal College of Surgeons of England.

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    inclined to the belief that sufficiently frequent exam- inations would greatly increase the proportion of cases in which this condition is found. Still more convincing as to the angioneurotic character of the renal paroxysms is the case about to be narrated. Severe renal symptoms initiated a series of typical Graves’s phenomena, ;n a woman whose mother and child also exhibit characteristic signs of vasomotor ataxia; but for nearly three yeais there has been no recurrence of renal phe- nomena, and frequent examinations of the urine reveal only occasionally a trace of albumin or a few shadow cells; while the patient has steadily put on flesh and recovered her full strength. Case XV.—Typical Graves’s syndrome, following sup- pression of urine with fever and delirium; recovery; hives; scanty perspiration; dilated pupils; intermittent ptosis; fre- quent retraction of upper lids; epistaxis; tremor; dermogra- phy; purpura; nail signs; mitral regurgitation. Mrs. X., aged thirty-one years, of American birth and German de- scent, following an attack of bronchitis with apparently pleuritic pain in the right side, had suppression of urine, with dry skin, fever, oppression in breathing, rapid and feeble action of the heart, headache, and slight delirium. I saw her two days later, in consultation. Under the treatment already instituted, secretion of urine had been resumed, and the urine was found to contain a moderate quantity of albumin, with very few casts and a few blood cells. The most evident sign upon casual inspection as the patient lay in bed was the flushed face and the retraction of the upper lids. The pulse was of low tension, full, weak, and rapid. There was a faint systolic murmur. The pupils were dilated; there was fine tremor of the hands; dermography was intense; the eye lid “hitch” was readily elicited ; but the thyreoid gland was not enlarged. I saw the patient again two months later. This time there was marked exophthalmos and considerable bilateral enlarge- ment of the thyreoid gland. The pulse was 140. That the diagnosis of Grave’s syndrome previously made was cor- rect, was now quite evident. The urine was normal in quantity and quality, and has so continued during the thirty months that have elapsed since that time. Under