Inflammation: an introduction to the study of pathology.

  • Adami, J. George (John George), 1862-1926.
Date:
1907
Catalogue details

Licence: In copyright

Credit: Inflammation: an introduction to the study of pathology. Source: Wellcome Collection.

Provider: This material has been provided by Royal College of Physicians, London. The original may be consulted at Royal College of Physicians, London.

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    PT. II originating from the proliferation of the endothelial cells of lymph-spaces, lymphatics, vessels, and, in fact, all structures. He has observed their mitosis, and has demonstrated the migration of such proliferated endo- thelial cells into the adjoining connective tissue (Fig. 6). These observations were confirmed by Councilman in his studies upon keratitis (24). We have, indeed, to recognise the very active part played by the vascular endothelium in inflammation, its pronounced proliferative and phagocytic properties. As pointed out by Abbott, Pig. 6.—Large [cells of endothelial origin (macrophages of Metchnikoff) from centre of lymph-gland in typhoid fever which have actively taken up lymphocytes (2.) and red blood-corpuscles (r. b. c.).—Mallory. Nicholson, and myself (52), within ten minutes after inoculating the B. coli into the circulation, the endothelial cells of the liver capillaries are swollen and have taken up numerous bacilli (Fig. 7). Before our observations, Werigo (53) had demonstrated the process whereby hepatic endothelial cells give off pseudopodia, by which anthrax bacilli are seized and subsequently englobed. liecently Behring and Much (54) have called attention to the great phagocytic powers of the endocardial endothelium. As Beattie points out, in omental in- flammation the cells lining the veins become swollen