A contribution to the pathology of the coronary arteries of the heart : based upon an analysis of 238 cases in which the condition of these arteries was ascertained after death : with observations upon the relationship of disease of these vessels to sudden death and angina pectoris / by John Lindsay Steven, M.D., Physician and Lecturer on Clinical Medicine, Glasgow Royal Infirmary.

  • Steven, John Lindsay, 1859-1909.
Date:
[1898?]
Catalogue details

Licence: Public Domain Mark

Credit: A contribution to the pathology of the coronary arteries of the heart : based upon an analysis of 238 cases in which the condition of these arteries was ascertained after death : with observations upon the relationship of disease of these vessels to sudden death and angina pectoris / by John Lindsay Steven, M.D., Physician and Lecturer on Clinical Medicine, Glasgow Royal Infirmary. Source: Wellcome Collection.

Provider: This material has been provided by The University of Glasgow Library. The original may be consulted at The University of Glasgow Library.

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    This was clearly a case in which the primary disease was of the cardiac muscle, and in the production of this the diseased coronary arteries doubtless played a part. (2) A stone-cutter, aged 64. Suffered from cardiac pain (angina pectoris), palpitation, dyspnoea, and fainting fits. The symptoms had been present for a year. The heart was gener- ally dilated and hypertrophied, and weighed 214 ounces. The valves were normal and competent, and, beyond passive hyperaemia, there was no lesion of the viscera. There was moderate atheroma of the coronary arteries and of the aorta. Here also the disease was manifestly cardiac throughout. (3) A quarryman, aged 54. The symptoms were of five months’ duration in all. He had had one or two severe anginous attacks. The other symptoms were dyspnoea, weak- ness, dependent oedema, and signs of aortic obstruction and regurgitation. There was great hypertrophy of the left ventricle and slight atheroma of the coronary arteries. The cardiac condition was clearly secondary to interstitial nephritis. (4) A reed-maker, aged 48. Symptoms of chronic bron- chitis, with persistent pain between the shoulders. There was slight atheroma of the coronary arteries, with hypertrophy of the left ventricle, secondary to interstitial nephritis. (5) A lamplighter, aged 39. While in hospital he had repeated daily unmistakable attacks of angina pectoris, in one of which he died. He had suffered from attacks of angina for eleven months before his admission. On inspection, there was found almost complete obstruction of the orifice of the left coronary artery by the encroachment upon it of a thickened patch of atheroma extending from the aorta. There was also a limited pericardial adhesion over the surface of the right ventricle, and Ijrown atrophy of the muscle fibre. With the exception of a .slight interstitial nephritis the other organs were healthy. This was very clearly a case of angina pectoris, due to the left coronary artery being obstructed. (6) An engineer, aged 48. Had suffered from anginal pain for four months, with evidence of chronic renal disease as well. The coronary arteries were atheromatous and tortuous, but the heart was not enlarged, and the valves were normal. There \vas chronic interstitial nephritis.