Albuminuric retinitis with vascular changes : aneurysms on retinal arteries / by A. J. Ballatyne.

  • Ballantyne, Arthur James.
Date:
[1908]
Catalogue details

Licence: In copyright

Credit: Albuminuric retinitis with vascular changes : aneurysms on retinal arteries / by A. J. Ballatyne. Source: Wellcome Collection.

Provider: This material has been provided by UCL Library Services. The original may be consulted at UCL (University College London)

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    in places unduly tortuous, and the following evidences of arteriosclerosis are present:— 1. Irregularities of calibre. 2. Silver wire, or copper wire, appearance. 3. Constriction of the underlying veins. 4. The first branch of the lower temporal artery, in its course towards the temporal side, shows at one point a sudden loss of its normal double contour, and a little further on changes into a bright white band of equal width, which is rendered more visible by contrast with a large haemorrhage across which it passes. Again, for a short distance the blood-column becomes visible, and, finally, its terminal branches may be traced as a number of fine white lines. 5. The main trunk of the lower temporal artery, at a point almost vertically below the macula, presents on one curve four separate aneurysms. These take the form of small globular dilatations of the whole lumen of the vessel. One of them is partly surrounded by a small retinal haemorrhage. The same vessel, far out to the temporal side, gives off a branch in the form of a narrow glistening white line. The veins are for the most part unduly wide and dark, with a well-defined central streak. As already stated, they are constricted where crossed by the arteries. The other changes present are round and flame-shaped haemorrhages of various sizes, irregularwhite soft-edged patches of exudate, and minute glistening spots scattered over the posterior part of the fundus. > The changes in the left eye were of the same kind, but the glistening degenerative spots were more numerous, and no aneurysms were found. The patient's general condition improved steadily, and lie was dismissed in July feeling well, with still a trace of albumen in the urine. The little aneurysms were already becoming less prominent, while the other evidences of vascular disease were increasing. , , He was next seen on 26th September. He had taken a long holiday, and had been back to work for five weeks, and was feelinff very well. The urine was practically as before and the bfood-pressure still high. (Systolic, 230 mm.; diastolic, ^IpwLlmoscopic examination -The hemorrhages» and exudative patches had almost entirely disappeared, even o-listenin^ degenerative spots were tewer. Iwo ot the torn aneurysms had disappeared. At the same time there wen evfSTthat the vascular disease was advancing. The branch